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Conversely, the mice displayed normal behaviour to noxious heat and non-noxious tactile stimuli.
All engrafted W/BF1 mice displayed normal immunologic competence 100 days posttransplantation.
In mice fed with chow, islets of the Elovl6−/− mice displayed normal architecture and β-cell mass compared with those of the wild-type mice.
Indeed, although defeated LDTgChAT-hM4 mice treated with saline showed strong social avoidance and anhedonic responses to sucrose solution, CNO-treated LDTgChAT-hM4 mice displayed normal social interaction and sucrose intake (Fig. 3b and Supplementary Fig. 5b, respectively).
Week 1 bleomycin-exposed mice displayed normal shifts in MLA with inspiration, suggesting normal lung recruitment despite significant radiographic and histological changes.
Similarly, hippocampal CA1 pyramidal cells in mutant mice displayed normal place-related activity in a full-cue environment but showed a reduction in activity upon partial cue removal.
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Glucose intolerant NAS-drinking mice display normal insulin levels and tolerance.
Engineered tissues composed of microvessels from diabetic mice display normal PVC coverage and Jagged1/Notch3 gene expression when implanted into non-diabetic hosts.
The H11 ES cell line gave rise to mice displaying normal ocular physiology.
G2019S-LRRK2 mice display normal locomotor activity in the open field at 6 and 15 months of age (Figure 5A B).
Indeed, Batf-/ deficient mice display normal Th1, and Th2 response but are deficient in Th17 differentiation, and are resistant to experimental autoimmune encephalitis[63].
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