Sentence examples for mice displayed mild from inspiring English sources

Both male and female transgenic mice displayed mild alterations in the circadian activity patterns, but no deficits in PPI.

TG mice with over-expression of Hand1-WT in heart did not display apparent phenotype by 32 weeks, which was in consistence with a recent report showing that Hand1-WT TG mice displayed mild hypertrophy but were predisposed to cardiac arrhythmia [31].

REDD1 KO mice displayed mild epidermal hyperplasia and slightly increased keratinocyte proliferation (Fig 3C and data not shown).

RIPCreα 2KO mice displayed mild glucose intolerance compared with littermate controls, but no reduction in peripheral insulin sensitivity.

Moreover, quadriceps nerves from Fig4 Floxed/plt, HB9-Cre mice displayed mild hypomyelination with increased g-ratio (the ratio between axon diameter and fibre diameter) at P30 (Fig.  1E and F; g-ratio: Fig4 Floxed/plt, HB9-Cre 0.71 ± 0.0004, 1189 fibres; controls Fig4 Fl/+ 0.68 ± 0.003, 1350 fibres; n = 4, P = 0.0057).

Compared to non-Tg mice, Tau4RTg2562 mice displayed mild TOC1 immunoreactivity in the CA1 region of the hippocampus (Fig.  4a,b), but TOC1 levels were nowhere near as robust as in another tau Tg mouse line (PS19) or in human AD hippocampus (Fig.  4, c and d, respectively).

GM3-deficient jck mice displayed milder PKD, revealing a pivotal role for ganglioside GM3.

Itoh and colleagues [ 19] showed in the model of antibody-induced arthritis that MMP-9 knockout mice displayed milder arthritis than their wild-type littermates.

The effects of caffeine were strongest in mice displaying mild cognitive impairment.

BAX-deficient mice display mild lymphoid hyperplasia, a minor increase in neurons in certain parts of the brain, and male sterility, but otherwise appear normal.

We report that Kv10.1-deficient mice display mild hyperactivity and longer lasting haloperidol-induced catalepsy in comparison to controls, but otherwise normal behaviour, and no obvious histological changes or differences in electrophysiological responses in the cerebellum.