Sentence examples for mice displayed markedly from inspiring English sources

Consistent with the increased activity of β-catenin, MUC1-CD transgenic mice displayed markedly increased mRNA levels of c-Myc, as well as elevated levels of cyclin D1 (Fig. 5C and D).

In contrast, wild-type mice displayed markedly reduced threshold levels of mechanical hypersensitivity after chronic opioid treatment (i.e. OIH), while Dcc heterozygous mice did not develop OIH.

In comparison to controls, LAQ-treated Rag1−/− mice displayed markedly reduced demyelination in the corpus callosum (Fig.  4a, b), fewer callosal microglia (Fig.  4c, d), fewer APP-positive axonal spheroids (Fig.  4e, f) and less fiber gliosis (Fig.  4g, h).

Consistent with previous reports from our group and others (Roos et al., 2013; Gioscia-Ryan et al., 2014), carotid arteries from aged mice displayed markedly impaired vascular relaxation in response to endothelium-dependent and -independent vasodilators compared to young mice.

TLR2/9 knocks out mice displayed markedly enhanced susceptibility to infection in association with combined defects in pro-inflammatory cytokine production in vitro, IFN-γ recall responses ex vivo, and altered pulmonary pathology [ 26].

Interestingly, the cells from PER2 S662G : Per2−/− mice displayed markedly attenuated levels of apoptosis that were similar to those of Per2− / − mice, and PER2 S662D : Per2−/− mice were even more resistant to X-ray-induced apoptosis than Per2 knockout (KO) mice.

Importantly, Gpr68-deficient mice display markedly impaired acute FMD and chronic flow-mediated outward remodeling in mesenteric arterioles.

Interestingly, diaphragms from mdx mice display markedly increased expression of CCR2 and its chemokine ligands and increased recruitment of Ly6Chi monocytes that differentiate into CD11bhi macrophages [273].

Grk6-null mice display markedly reduced neutrophil mobilization from the bone marrow to the peripheral blood in response to G-CSF in spite of having normal hematopoiesis.

Tryptophan hydroxylase-1 is the chief enzyme responsible for synthesis of peripheral 5-HT [12], and Tph1 mice display markedly reduced concentrations of 5-HT in whole blood (92% decrease), platelets (90% decrease) and plasma (80% decrease) [13], [14], [15].

MUC1-CD transgenic mice display markedly increased levels of c-Myc and cyclin D1 in their mammary tissues at virgin and pregnancy stages, suggesting that induction of mammary hyperplasia by MUC1-CD likely occurs through activation of β-catenin and subsequent upregulation of c-Myc and cyclin D1 gene expression in the mouse mammary gland.