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Compared to WT animals, HO-2 null mice displayed an increase in both visceral and subcutaneous fat content and reduced circulating adiponectin levels.
Drug-treated mice displayed an increase in influenza nucleoprotein-specific cytotoxic T cell activity.
In addition, GRK3 -/- mice displayed an altered HR recovery from MCh-induced bradycardia.
Following sepsis induction, however, HDC−/− mice displayed an evidently lower levels of those cytokines compared with WT mice.
Instead, ezrin−/− mice displayed an increased hematocrit, platelet and red blood cell (RBC) counts (Table 1).
Indeed, SF1 gene knockout mice displayed an abnormal VMH development leading to obesity [23].
As previously shown, HP mice displayed an improvement in glucose tolerance [11].
In summary aged mice displayed an attenuated response to respiratory F. novicida infections.
In this regard, it is noteworthy that CAG200 mice displayed an energy metabolism defect that is shared by HD.
Moreover, CNTF −/− mice displayed an increase in axonal structure proteins like alpha-internexin indicating an enhanced axonal disintegration.
As expected IL-15Tg mice displayed an increased percentage of NK cells compared to wild type control mice (Fig 3A).
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