Sentence examples for mice displayed abnormal from inspiring English sources

To investigate whether En2−/− mice displayed abnormal behavior relevant to ASD, they were monitored in tasks designed to assess social maturation as well as learning and memory.

Behavioral analyses revealed paternally duplicated (patDp/+) mice displayed abnormal behaviors resembling the symptoms of ASDs.

RhoE deficient mice displayed abnormal motor patterns and motor behavior (Video S1).

Interestingly, Sei1-null mice displayed abnormal insulin secretion and blood glucose clearance, as well as a reduction in pancreatic β-cell surface.

In addition, the mice displayed abnormal structures at the nodes of Ranvier, leading to electrophysiological defects characterized by reduced conduction velocities in motor nerves (Susuki et al., 2007).

We previously demonstrated that global heterozygous Bmpr1a-deleted mice displayed abnormal glucose homeostasis, but the role of other insulin-sensitive tissues including the liver, skeletal muscle and adipose tissue could not excluded (Scott et al., 2009).

Thus, TSP-2-null mice display abnormal connective tissue architecture and increased angiogenesis in the dermis, and heal wounds at an accelerated rate.

Isoform-specific Shank3 e4-9) homozygous mutant mice display abnormal social behaviors, communication patterns, repetitive behaviors and learning and memory.

Mutations in Drosophila Ago1 cause a severe loss of all types of neurons and glial cells while dicer deficient mice display abnormal neurogenesis and gliogenesis in the developing central nervous system (Kataoka et al., 2001; Kawase-Koga et al., 2009).

Taken together, these results show that Pink1−/− mice display abnormal brain cytokine expression in response to peripheral LPS injection and suggest that Pink1−/− mice may be more susceptible to inflammation-induced DA neuron death [64], [65], [66], [67], [68].

We found that these mice display abnormal synapse organization and maturation in vitro, altered synapse density in the adult brain, enhanced glutamatergic transmission and reduced synaptic plasticity in CA1 hippocampus.