Exact(2)
(A) Representative ECoG recordings of WT, GluN2C HET, and GluN2C KO mice display typical awake responses.
In particular, homozygous SSADH knock-out (SSADH−/−) mice display typical absence seizures that appear at the beginning of the third postnatal week, evolve into myoclonic and generalized convulsive seizures, and finally progress to lethal status epilepticus [6], [8], [9].
Similar(6)
Over time, db/db mice displayed typical phenotypes of obesity, polyuria and hyperglycemia.
The freshly isolated mouse hepatocytes display typical cubic, nonproliferating morphology 2 hr after being plated (Fig. 2A).
Indeed, Znt7-mutant mice do not display typical zinc deficiency symptoms, and their phenotypes cannot be rescued by zinc supplementation [ 33].
Vertebrate aromatase genes display typical putative structural domains.
Correspondingly, these mice display clinical signs typical for motor neuron disease, namely a gradual development of motor deficit followed by pareses and paralyses, which are most obvious in limb muscles.
The mice displayed a typical circadian rhythm having a higher RR interval and a higher HRV (all HRV parameters) during light period when the vagal regulation is expected to be higher.
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