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In preliminary studies, we observed that virus strains of low (BJx109, H3N2), intermediate (HKx31, H3N2) or high virulence (PR8, H1N1) in mice differed markedly in their ability to recruit neutrophils to the airways.
This vascular PrPres distribution in tg44 mice differed markedly from the pattern seen in non-transgenic C57 mice expressing anchored PrP, where PrPres was not in a perivascular location, but instead tended to cluster around parenchymal cells with large nuclei, possibly neurons or astroglia, as has been reported previously [ 34].
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DBA2 mice differ markedly in the expression levels of their hepcidin genes when compared with Balb/c and C57BL/6 mice.
NZB and NZW parental strains express the distinct P2X7-L and P2X7-P alleles of P2RX7, respectively, while lymphocytes from these and (NZB × NZW F1 mice differ markedly in their responses to P2X7 receptor stimulation.
The two mouse strains differed markedly in basal anxiety level.
In this regard, mouse cells differed markedly from human cells.
When control mice were studied, the profile of fractalkine cleavage differed markedly from that in NOD mice.
Gut bacterial numbers and phylum abundance were similar in mice containing mouse and human microbiota, but bacterial species differed markedly (Chung et al. 2012).
The second phase of anaemia development differed markedly between the three mouse strains (and in previous studies between cattle breeds [3], [4], [5], [6], implying that this mechanism is dependent on genetic and host background.
Furthermore, the differential of the BALF cells differed markedly as the DHA-pretreated mice had significantly less neutrophils and lymphocytes beginning on day 7 and continuing through day 21.
The acceptability of surveillance differed markedly between countries.
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