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These mice differed from each other by interchanging their H-2 haplotypes in two different genetic backgrounds.
The sensitivities against psychostimulants in orexin/ataxin-3 mice differed from those in orexin KO mice.
In these conditions, the behaviour of TRαAMI/Cre3 mice differed from that of control littermates only in the light-dark box.
The results of the transmission study in TgBoPrP mice differed from those obtained in the case of wild-type mice.
Immunological analysis showed that I/St mice differed from less susceptible A/Sn mice in that they exhibited lower antimycobacterial responses [11], [17] and more prominent inflammatory reactions [11], [16], [18].
We have noted that Ckb is more highly expressed by CD4SP thymocytes than CD8SP thymocytes, whether Ckb participates in regulating CD4/CD8 lineage decision is not directly addressed here, however, the CD4SP/CD8SP ratio in CkbTg mice differed from that in littermate mice (Figure 3B), which indicates altered lineage commitment.
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In addition to obvious differences such as small size and short lifespan, mice differ from humans in a range of anatomical, physiological, energetic, electrophysical, and mechanical properties that include heart rate, coronary artery structure, and contraction/relaxation kinetics.
Surprisingly these mice differ from Mc1r mutant mice as they have a pain response indistinguishable from mice with a functional Mc1r.
In order to assess how jnk1−/− mice differ from wild-type mice we analyzed gene expression patterns prior to and following mechanical ventilation.
In a key paper, it was reported that the gut microbiota in obese humans and mice differ from those in lean individuals [9].
Though we observed a slight but significant reduction in the percentage of activated microglia and astrocytes at onset stage, neither survival nor motor performance of double transgenic mice differ from those of single transgenic ALS mice (Figure 4 5).
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