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The MuSK-inactivated mice develop myasthenic symptoms and die prematurely due to severe muscle weakness.
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Supplementary Figure 6 Pik3cdE1020K/+ mice develop IgM autoantibodies.
Aged Pten-ΔTreg mice develop non-malignant TH1 inflammation.
Concurrently, mice develop delayed deficits in LTP and cognition.
Bad-deficient mice develop diffuse large B cell lymphoma.
Both NOS1-/ and NOS3-/ mice develop age-related hypertrophy, although only NOS3-/ mice are hypertensive.
The mice developed F.F.I.
The normal mice developed osteoarthritis as expected.
There was no significant difference between Krt76+/+ and Krt76+/− mice; however, Krt76−/− mice developed OSCC earlier.
DePinho said none of Harvard's mice developed cancer after the treatment.
129/SvEv mice developed more severe pathologic changes in spleen and bone marrow, whereas C57BL/6J mice developed more severe renal pathology.
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