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Although STAT5 promotes survival of hematopoietic progenitors, STAT5−/− mice develop mild neutrophilia.
Surprisingly, however, STAT5−/− mice develop mild neutrophilia rather that neutropenia [23], [27].
In agreement with its role as a co-chaperone, Dnajc3−/− mice develop mild diabetes.
In the absence of the MRL abnormalities and on the C57/B6 background, these mice develop mild autoimmune disease.
Neither of the parent strains develops overt pathology, although NZB mice develop mild autoimmune hemolytic anemia (Sang et al., 2012).
At ambient temperatures below thermoneutrality mice develop mild hypothermia at intermediary doses of LPS and excessive hypothermia at high doses, indicative of a septic shock-like condition (Krakauer et al., 2010).
Similar(53)
Wildtype (WT) mice developed mild peribronchial inflammation (Figure 1A).
The challenged mice developed mild clinical symptoms (reduced activity) until about six days PI, after which time they recovered (data not shown).
Importantly, C. jejuni associated IL-10−/−; NF-κBEGFP mice developed mild (day 5) and severe (day 14) ulcerating colonic inflammation and bloody diarrhea as assessed by colonoscopy and histological analysis.
In the small intestine, only 1/8 mice developed mild inflammation with irregular crypt architecture, 10 25% goblet cell loss, increased leukocytes in the lamina propria and slightly increased levels of polymorphonuclear cells in the lamina propria.
A minority of older MRL-lpr/lpr mice developed mild arthritis [ 21].
More suggestions(15)
mice develop extensive
mice develop early-onset
mice develop high
mice develop local
mice develop glaucoma-like
mice develop severe
mice develop spontaneous
mice develop pituitary
mice develop renal
mice develop progressive
mice develop hepatic
mice develop significant
mice develop thymic
mice develop systemic
mice develop α-synuclein-dependent
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