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Ex vivo, aortae excised from Fkbpl+/+ mice demonstrated ordered sprouting; however, this process appeared to be significantly enhanced in Fkbpl+/neo, and vessel sprouts were highly branched and numerous.
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Saline-treated PAI-1−/− mice demonstrated significantly less IL-13 than similarly treated WT mice (p < 0.05).
Further, S. pneumoniae infected-PAI-1−/− mice demonstrated significantly higher plasmin activity than infected WT mice (p = 0.04).
S. pneumoniae-infected PAI-1−/− mice demonstrated significantly higher plasmin activity than identically treated WT mice (p = 0.04).
C57BL/6 mice demonstrated a distinct profile of microbiome file in gut.
By morphometry, S. pneumoniae infected mice demonstrated significantly more pleural thickening than saline controls (p < 0.001).
All mice demonstrated full visual capability.
Three mice demonstrated subarachnoidal hemorrhages.
All mice demonstrated hyperactivity during housing.
GRHL3 -/- mice demonstrated defects in cell migration.
As the BALB/c and the IFNAR-/ 129SvEv mice demonstrated very similar viral loads in their brains, we also compared their gene expression profiles upon infection with MHV in order to identify type I IFN-dependent transcriptional responses.
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