Sentence examples for mice could induce from inspiring English sources

Here, we tested if systemic injection of Pam2 lipopeptides in mice could induce an effective anti-tumor immune response.

Thus, immunization in MD4 mice could induce antibody production primarily from these sources, which would not be a major contributing factor in mice with a polyclonal repertoire.

Indeed, blocking HMBG1 with small interfering RNA in a murine model of liver fibrosis inhibited collagen production [ 133], whereas the injection of recombinant HMGB1 into mice could induce lung pathologies similar to that observed in cystic fibrosis [ 132].

More recently, it was reported that intracerebral injection of extracts from sick A53T human α-synuclein trans genic mice (line M83) into younger healthy M83 transgenic mice could induce disease [ 50, 51].

To evaluate if autoreactive CD4+ T cells from scurfy mice could induce autoantibody production via T cell-mediated B cell help, we transferred purified CD4+ T cells from scurfy mice and WT controls into B6/nude mice, which completely lack CD4+ T cells but possess a normal B cell repertoire.

Experimentally it was reported that the intracerebral injection of extracts from moribund A53T human αS transgenic (Tg) mice (line M83) that develop a late onset severe motor phenotype associated with widespread formation of neuronal αS inclusions into younger healthy M83 Tg mice could induce these cellular and phenotypic pathologies [ 17- 19].

When astrocytes derived from SOD1 glial progenitors were transplanted into mice, they could induce host motor neuron death, focal weakness of the corresponding limb, and gliosis of host astrocytes and microglia [ 67, 68].

Besides that, the mouse tumors could induce γH2AX formation in highly proliferative tissues distant from implanted tumors.

We found that epitopes from both human and mouse heparanase could induce an effective antitumor immune response.

This suggests that the high overexpression of expPABPN1 in the OPMD mouse model could induce similar molecular changes as in normal muscle aging.

In a transgenic mice model, CHD1L could induce spontaneous liver tumors formation (Chen et al., 2009b).