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Together these data suggest that enhanced c-Kit expression in 4T1 bearing mice correlates with an altered metabolic state in peripheral neutrophil populations where mitochondrial metabolism is increased due to increases in fatty acid utilisation required for maintenance of NADPH levels.
The novel oocyte/ovary phenotype of the Prep1i/i mice correlates with the increased production of Hoxb4.
Enhanced neurogenesis in mice correlates with all known antidepressant treatments including exercise, medications, and electroconvulsive therapy [2].
This suggests that this phenotype, both in humans and mice, correlates with an impaired ability to control lung pathology.
This behavioral deficit, specific to the D36 mice, correlates with their deficiency in both hippocampal LTP and LTD when compared with either WT or KO mice.
For instance, severe combined immunodeficiency (SCID) in mice correlates with a nonsense mutation within the gene of the DNA-PK catalytic subunit (DNA-PKcs) [13] [15].
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Our most surprising result was that the protection in mice correlated with the level of antibody binding on the arrays.
Neutralization of lethality in mice correlated with neutralization of in vitro coagulant activity in human plasma.
Results: The degree of structural and behavioral abnormalities in adult mice correlated with the duration of hypoxia ischemia on P10.
Instead the cancer susceptibility of Krt76−/− mice correlated with a higher number of Tregs in secondary lymphoid organs and an enhanced accumulation of Tregs in the tongue and squamous stomach, which increased further in the tumour microenvironment (Fig. 6e h).
Overall, increased resistance to influenza challenge in the mice correlated with an enhanced Th1-type M2e-specific antibody response induced by vaccination.
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