Sentence examples for mice caused increased from inspiring English sources
Another study [44] showed that high-dose PTX treatment in PyMT mice caused increased macrophage infiltration that protected tumors from cell death and facilitated tumor progression and metastasis.
In mouse, conditional Atp6ap2 deletion in glutamatergic neurons (Atp6ap2 Camicecausedmincreaseded increaspontaneouseous locomotor activity and altered fear memory.
Synovial Nlrp3 and Casp1 expression further correlated with Acp5 (encoding TRAP), while neutralization of IL-10 receptor signaling in control mice caused increased expression of Nlrp3 and Casp1.
Comparable to genetic ablation of Alox15, pharmacological blockade of 12/15-LO 12/15-LOce caused increased CXCL1 plasma levels and decreased CXCR2 expression on circulating neutrophils compared to vehicle-treated control mice under baseline conditions and after LPS exposure.
In line with an anti-inflammatory role for IL-10 in experimental arthritis, AIA in IL-10KO mice caused increased joint swelling and an overall exacerbation of joint pathology as compared to wild-type (WT) controls.
At early ages, F66 transgene expression in Dysf −/− mice (i.e. F66 Dysf −/− mice) caused increased muscle mass to a similar extent as F66 did in a wild-type (wt) background (Fig. 1A D; Supplementary Material, Table S1).
Here we have tested the hypothesis that disruption of Nrf2 in mice causes increased atherosclerosis.
Insulin depletion by STZ administration in six months-old non-transgenic mice causes increased tau phosphorylation, without its deposition or NFT formation.
Pubertal exposures in male mice cause increased body weight gain, hepatic steatosis, hyperinsulinemia, and hyperleptinemia (Zuo et al. 2011).
Conversely, overexpression of GM-CSF in genetically modified (i.e., transgenic) mice causes increased lung size, excessive growth (i.e., hyperplasia) of alveolar epithelial cells, and improved surfactant protein removal from the alveolar space (Ikegami et al. 1997).
Targeted disruption of the MEPE gene in mouse causes increased bone formation and bone mass, suggesting that MEPE plays an inhibitory role in bone formation and mineralization [ 15].
