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Among the tissues examined in PLP mice, brain, lung and thymus showed no reduction in weights as compared to control tissues.
Figure 3 referred to the mean value of SOD inhibition activities in Tx and Rx for mice brain, lung, and liver tissues.
The results obtained from the experimental analysis of MDA levels in mice brain, lung, and liver tissues are presented in Figure 1.
In liver tissue, there was a significant difference between Tx (68.50 ± 1.82%) and Rx (59.13 ± 2.0%) with P = 0.04. Figure 4 shows the levels of GSH content in mice brain, lung, and liver tissues.
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VEGF binding activity was blocked by 6a-P in embryonic mouse brain, lung and kidney (Supplemental Figure S2).
Interestingly, cells and tissues from p27 null mice, including brain, lung, and retina, present an elevated basal expression of Sox2, suggesting that p27 contributes to the repression of Sox2.
On the contrary, the results obtained on thin sections of unirradiated mice tissues including brain, lung, heart, and small intestine have shown that they are almost completely negative for γH2AX (Rübe et al. 2008).
Similarly, total RNA was isolated from mouse brain, muscle, lung, spleen, heart, liver, kidney, and testis.
Ltbp3 transcripts were also present in adult mouse tissues in heart, brain, lung and kidney (7, 8).
Following the systemic injection of monocytes/macrophages containing the fluorescently labeled microspheres, mice were sacrificed and the brain, lung, liver, and the heart were dissected.
At the end of the experiment, animals were weighed and all mice were euthanized, and tumours, brain, lung, heart, liver, spleen, intestine and kidney tissues and serum were stored at -80°C.
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