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Male C57BL/6 mice at age of 8 12 weeks were purchased from the Shanghai Laboratory Animal Company SLACC, Shanghai, China).
Wild-type mice and low-density lipoprotein receptor gene deficient (LDLR−/−) mice at age of 8 weeks were placed on either control diet or high-fat diet (HFD) for 12 weeks.
The hematopoiesis of wild-type (WT), Gadd45a−/−, ATM−/−, and ATM−/− Gadd45a−/− mice at age of 2 3 months was analyzed by FACS.
The number of HSCs in WT, Gadd45a−/−, ATM−/−, and ATM−/− Gadd45a−/− mice at age of 2 3 months were analyzed by FACS.
Cage libraries were prepared as in [10], using the visual cortex of mice at age P21, P26, P54 and P71.
Consistent with the normal quantity of cTnT observed, no phenotype abnormalities were observed in Tnnt2+/− mice at age 8 10 weeks.
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Heads were obtained from euthanized wild type and K14-Cre p120-cKO mice at ages 3, 7, 9, and 15 days.
In the present studies, we used mice at ages 5, 12 and 28 months of age.
Electrophysiological measurements were performed in SMNΔ7 and control mice at ages 3 13 days.
Eight-week-old CD98Δ/+ female mice were mated with CD98Δ/+ male mice at ages of 8-16 weeks to obtain homozygous mutant (CD98Δ/Δ) mice.
In the present study, we used the MT42 line of TGFα-expressing TG male mice and CD1 wild type (WT) male mice at ages 6 to 8 weeks.
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