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However, myeloid/haematopoietic cell-specific TLR4 knockout mice as generated by adoptive BM transfer were not protected from HFD-induced skeletal muscle insulin resistance, whereas hepatic insulin sensitivity was significantly improved (supplementary Fig 19).
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Erk1-knockout mice were generated as described.
Stat3 f/f mice were generated as described.
Bmpr1a− /fx -; Mox2Cre+/− mice were generated as described elsewhere (Miura et al., 2006).
Mice with human APOE genes replacing murine APOE genes (henceforth described as 'APOE mice') were generated as published [78], [79], [80].
B6J-TRAF6−/− mice [47] and B6N-IL-33−/− mice [48] were generated as described elsewhere.
Lyn −/− Csk AS mice were generated by crossing Csk AS and Lyn −/− mice.
CSRNP-deficient mice were generated essentially as previously described [33].
ICOS−/− mice were generated as previously described [12] and backcrossed 8 generations on the C57BL/6 background.
15 Pxr−/− mice were generated as described 17 and Car−/− mice were generously provided by Dr. Masahiko Negishi (NIEHS).
RIP3 knockout mice were generated as described previously [ 11] and crossed to C57BL/6 mice for ten generations.
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