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These results clearly indicated that human B cells developed by DRAG mice are fully functional.
Furthermore, our results demonstrated that DCs from Jak3−/− mice are fully capable of leaving the epidermis.
Approximately equal numbers of wildtype and heterozygous Sdhd mice were obtained, indicating that these mice are fully viable (Table 1).
Thus, when mice are fully trained in the 5C-CPT, performance did not differ significantly between strains, although several trends were evident.
Importantly, several cytokines in these mice are fully repressed by GC treatment while regulation of many genes involved in metabolic side effects is abolished [8], [9].
However, because AID-deficient mice are fully fertile, the molecule is not essential for meiosis.
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Immunized mice were fully protected against lethal doses of virus challenge.
Consistently, IP infected mice were fully protected in a challenge with 104 oral S. Typhimurium, while protection was partial in orally immunized mice.
Although pulmonary (CD11c+) cells from old mice were fully capable of producing cytokines in response to M. tb infection, we demonstrate that in contrast to young mice, M. tb induced cytokine production occurred independently of TLR-2.
Immunized mice were fully protected against a homologous challenge with a lethal dose of BTV-4 and partially cross-protected against a heterologous challenge with a lethal dose of BTV-1.
Blood neutrophils from STAT5−/− mice were fully differentiated.
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