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AM from MCP-1−/− mice also demonstrated less superoxide and impaired phagocytosis over the controls.
Balb/c mice responded similarly, although SH operated mice also demonstrated a slight lowering of hematocrit (Fig 4, right).
Examination of LTD in 2 wk old mice also demonstrated a substantial deficit in the D36 mice.
Vipr2−/− mice also demonstrated more stage shifts between wake, NREM and REM sleep and more arousals than WT mice.
Measurement of Rpn13-associated enzyme activities in KO mice also demonstrated differential, tissue-specific effects of Rpn13 deletion.
An in vivo study using C57BL/6 mice also demonstrated the anti-inflammatory properties against LPS in peritoneal macrophages.
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ERα KO mice also demonstrate lower levels of physical activity [44].
The results from the fat-1 mice also demonstrate that major alterations in mitochondrial ROS production and ETC enzyme activities can occur with fatty acids changes in PE and PC, and without major fatty acid changes in CL.
This deficit is primarily driven by AC1, as AC1-deficient, but not AC8-deficient mice also demonstrate significant reductions in phosphorylation of synapsin and eEF-2 in cortical and hippocampal tissues.
During embryonic development and the early post-natal period, mice also demonstrate a remarkable regenerative capacity.
Interestingly, MMTV-Wnt1 mice also demonstrate increased Sca1 expression in the mammary gland [ 28, 29].
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