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Hence the deleterious effect of CCBs in Marfan mice also appears to be AT1R-dependent.
Autoimmune arthritis in SKG mice also appears to be highly dependent on the CD4+ T cells secreting IL-17 [ 47].
Further, the enhanced Th2 response in Lyn −/− mice also appears to be contributed to by the lack of Lyn signals through the FcϵRI in basophils [ 61].
A dyslipidemic state, as found in apoE knockout mice, also appears to include age-dependent accumulated oxidized DNA base lesions in the liver (Folkmann et al. 2007).
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The mutant mice also appear to have weaker memories of scary encounters than normal mice do.
Interestingly, female mice also appeared to respond better to AV.RSV.MCAT treatment.
Male mice also appeared to die over a greater time span (Figure 3D).
DBA mice also appear susceptible to increased body weight when provided a HF diet.
The differences between the immune response in humans and mice also appear to apply to the downstream effectors of IFN-γ, NOS2 and IRG.
Protected mice also appeared to have increased CD8+ T cells in lung granulomatous regions at day 7 post-secondary challenge (Figure 7E) compared to non-protected mice (Figure 7B).
The UR-KD mice also appeared healthy throughout the study, but displayed oily fur and stool discoloration (yellowish).
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