Sentence examples for mice a well from inspiring English sources
Interleukin-10 deficient mice, a well characterized experimental model of inflammatory bowel disease, spontaneously developed a Th1 T cell-mediated colitis with many similarities to Crohn's disease.
The aim of this study was to examine the factors that contribute to energy imbalance in the GH releasing hormone knock out (GHRHKO) mice, a well established model of GHD.
Objective: To investigate the expression of a novel member of the mannose receptor family, Endo180 (also known as uPARAP), and the distribution of Endo180 ligand(s) in the articular cartilage and growth plate of normal CBA mice and STR/ort mice, a well characterized model of spontaneous osteoarthritis.
In 35-day-old control mice a well developed SC barrier preventing passage of lanthanum was seen in all tubules analyzed (Figure 2G).
We here confirm and further extend these findings by demonstrating that VASP also prevents BBB damage and edema formation in the brain after tMCAO in mice, a well established in vivo model of ischemic stroke.
To further optimize this animal model, we tested other conditions for SS disease induction using the same peptide, and switched to SJL/J mice, a well established autoimmune prone animal model [9], [10].
The aim of this study was to evaluate the effect of intrapyloric transplantation of neural stem cells (NSCs) on gastric emptying and pyloric function in nNOS−/− mice, a well-established genetic model of gastroparesis.
ApoE KO mice, a well-established animal model of atherosclerosis, had higher expressions of KLF14 in aorta tissues than that in C57BL/6 J mice when fed the high-fat diet (HFD) or standard chow diet.
The objective of this study was to investigate if sleep wake patterns are altered in 5XFAD mice, a well-characterized double transgenic mouse model of AD which exhibits an early onset of robust AD pathology and memory deficits.
Therefore, we examined the extent to how VSL#3 reduced atherosclerosis, the cardiovascular inflammation, and the effects of VSL#3 on the gut satiety hormones, inflammatory profile and the changes in the gut microbial community in apolipoprotein E knockout (ApoE−/−) mice, a well-established and popular model for atherosclerosis (Meir and Leitersdorf 2004).
To address this issue, we employed a strain of RARE-hsp68-lacZ transgenic mice, a well-established mouse model of a C57BL/6 genetic background, to detect endogenous RA activity [15].
