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In the tissue slices from young mice, a decrease in long-term potentiation (LTP) was observed.
On the other hand, in the homozygous mice a decrease was seen in the medial NTS and AP.
Significant responses of the glucose level to isoflurane have been reported by other groups; an increase in the blood glucose level in the brain was observed for adult rats in [32, 33] after 60 to 75 min of isoflurane, while in neonatal mice a decrease in the glucose level was observed in [34].
In IL-17R deficient mice, a decrease in arthritis severity was characterized by increased synovial apoptosis, reduced proliferation and a marked reduction in synoviolin expression.
The present study suggests two mechanisms to account for the compromised muscle strength in rpS6P−/− mice: a decrease in total muscle mass, reflecting diminished CSA of myofibers, and reduction in readily available energy.
In mice, a decrease of BP parameters to lower values compared to a KX anaesthesia was reported [ 56].
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In eNOS-/ mice, a decreased iNOS expression was observed compared with septic WT mice.
There was increased expression of glomerular FKN and p65 protein in LPS-induced mice (P < 0.01 and P < 0.05 respectively) when compared to control mice, a decreased expression in MP-treated mice compared to control mice (P < 0.05) and a decreased expression in LPS + MP and LPS + SC mice compared to LPS-induced mice (P < 0.01 and P < 0.05 respectively).
Mutations in Zip1, Zip2, or Zip3 also confer on mice a decreased ability to survive under dietary zinc limitation, particularly during pregnancy, when zinc absorption is normally increased [ 27- 29].
In the mouse, a decrease in the efficiency of DNA repair with age has been observed (37– 37).
Microglia from old PS1-APP mice, but not from younger mice, have a decrease capacity to clear Aβ.
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