Sentence examples for mg toxicity from inspiring English sources

MG is formed as a by-product of glycolysis and MG toxicity results from its damaging capability leading to modifications of proteins, lipids and nucleic acids.

We demonstrate the importance of macromolecule catabolism for resistance to MG, confirming and extending known mechanisms of MG toxicity.

Specifically, we demonstrate the importance of macromolecule catabolism processes for resistance to MG, confirming and extending known mechanisms of MG toxicity, including modification of DNA, RNA, and proteins.

Drought-induced MG toxicity is evident in increased MG content of 90 and 107 %, respectively, after 24 and 48 h of drought stress.

Thus, exogenous GSH supplementation with drought significantly enhanced the antioxidant components and successively reduced oxidative damage, and GSH up-regulated the glyoxalase system and reduced MG toxicity, which played a significant role in improving the physiological features and drought tolerance.

But exogenous GSH enhanced components of the antioxidant system in drought-affected mung bean seedlings, which alleviated oxidative damage, up-regulated the glyoxalase system, reduced MG toxicity, and modulated the proline and water content, contributing to drought tolerance.

Thus, the results would suggest the use of organisms >5 mg for toxicity tests.

At the daily dose of 250 mg, skin toxicity and diarrhoea are the main gefitinib side effects, occurring in 46.6 and 39.8% of cases, respectively (Fukuoka et al, 2003).

An important defence against MG-related toxicity is the glyoxalase complex (formed from GLO-1 and glyoxalase II components), which converts MG to d-lactate using GSH as a co-factor [ 31].

MG-132 toxicity resulted in an increase in omi gene expression at 12 hours post-treatment.

In Mdr1ab/Mrp1 TKO mice receiving 0.5 mg kg−1 toxicities manifested by arrested mitoses were found in the intestines, skin, bone marrow, the adrenal medulla and in the bases of the incisors.