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RESEARCH DESIGN AND METHODS: We show that glucose, acting through oxidative stress, activates the transcription factor Foxo1 in vascular endothelial cells.
Using difference-in-difference methods, we show that this had few of the negative effects claimed by critics.
Using biochemical and structural probing methods, we show that despite sequence variability they contain a common three-dimensional fold.
Moreover, differently from the existing methods, we show that the presented LMIs are always feasible for small enough delays.
Using quasi-experimental methods, we show that the policy reform has strikingly reduced the prices in the wholesale market.
Combining analytical and numerical methods, we show that the 1D responses of grid cells with stable 1D fields are consistent with a linear slice through a 2D triangular lattice.
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Comparing our algorithm with other methods, we showed that the new approach is able to solve some of the previous method's drawbacks.
By biochemical methods we showed that these antibodies were specific for denatured SOD1.
As opposed to previous methods, we showed how to extend the resolution of nucleosome calls beyond that of the measurements.
Using phylogenetic methods we showed that there are low levels of recombination between similar modules in functionally different NRPS gene clusters within one and the same strain.
By gene transfection methods, we showed that C/EBPα gene could inhibit the growth of HeLa cells and reduce the invasion of HeLa cells.
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