The scientific method can explain the world around us in an elegant and creative way, but scientists cannot escape the influence of external pressures and personal bias.
Our method can explain why the image screw dislocation is required.
However, low expression levels of the respective transcript and the limited sensitivity of the experimental method can explain failed detection of the restricted expression pattern.
They are calculated from the structures of reactant molecules utilizing quantum-chemical methods, which can explain reactivity correlated with the activation barriers of a complete molecule as well as of molecular fragments and substituents [37, 38].
Though Huang and colleagues have presented the first attempt at computationally modeling protein stability, this work can be improved through the removal of potential experimental bias from the data, and the use of a computational method that can explain its predictions.
For example, GGEA needs in 73% of its top ranked gene sets a single additional gene to make the differentially expressed genes in a particular set connected, whereas the best set enrichment method, ORA1, can explain only 42% of the sets with a single addition gene (SAFE: 35%, GSEA: 34%, ORA2: 32%, SAMGS: 29%).
Application of the methods of science can explain the evolution of the universe, according to the known laws of physics and mathematics, from the Big Bang until recently.
Though differences in the signal measured by the 2 neuroimaging methods can potentially explain these different results, based on our current findings we propose an additional interpretation.
In their discussion of the Heinzle study, Raemaekers and colleagues noted a few differences in the methods and analyses, but ultimately concluded: "Whether this difference in methods can fully explain the difference in results is unclear…" We agree that there was considerable variability in the imaging and analytical approaches across all three studies (Heinzle's, Raemaekers', and ours).
Given the possibility that T IBj5) might be a complex rearrangement that includes a (I ; IV) RT (see Materials and Methods), homozygosity for the mutation can explain why T IBj5) Nta × T(B362i) NtA yielded only inviable ascospores (Table 5).
