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It has been demonstrated that a complex cascade of inflammatory responses may mediate alteration in metabolic processes, contributing to the development of cancer cachexia [ 5].
Other research indicates that air pollution exposure, with traffic as a major source in many cities, may operate through inflammatory pathways to initiate metabolic processes contributing to diabetes formation [ 18, 19].
Because these models aim at comprehensiveness, they are able to account for all main metabolic processes contributing to growth, i.e. the production of energy and biomass precursors from external metabolites.
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We speculate that genetic variations in basic metabolic processes contribute to their different tolerance of hypoxia, which is worth examining in the future.
Many of these targets are components of signalling pathways or metabolic processes that contribute to one of the hallmarks of the cancer phenotype.
It is perceivable that data on metabolic processes can contribute to the understanding of the pathophysiology of schizophrenia.
Disruption of these processes is known to affect diverse developmental and metabolic processes, and contributes to various diseases, including neurodegenerative diseases, diabetes, and cancer [ 2– 5].
High expression of genes enriched for metabolic processes may contribute to the rapid growth through the larval instars as individuals consume pollen provisions in their cells.
In addition, these metabolic processes may contribute to the formation of reactive oxygen species (ROS) [ 9], which are inactivated by anti-oxidant defense mechanisms [ 10].
Investigations related to molecular cascade interactions are providing insight into the genetic, signaling, bioenergetic, and metabolic processes that contribute to muscle health and disease.
Several metabolic processes may contribute to these low HDL levels: decrease in free cholesterol removal from cells, low cholesterol esterification rate and high cholesteryl ester transfer from HDL to apo-B containing lipoproteins [ 47].
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