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The above mentioned data indicate TLR3 and its downstream molecules in macrophage were activated by pristane, and then took part in the pathogenesis of PIA.
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Our above-mentioned data indicate that Thr538-Pro, which is adjacent to the WWP1 binding site (PPxY motif), is critical to Pin1-mediated regulation of p63 α stability.
The above-mentioned data indicate that changes in the Switch 1 loop specifically activate URA2, despite their adverse effects on growth and their reduced genome-wide RNA polymerase II occupancy.
The above-mentioned data indicate a correlation between RDW and other known atherosclerosis-predictive factors, although it is impossible to clearly identify the mechanism by which high erythrocyte anisocytosis serves as a negative prognostic marker in patients with CAD.
The above mentioned data clearly indicate the utmost importance of effective treatments for MDD.
As previously mentioned, the available data indicate that adjuvant chemotherapy with an anthracycline-containing regimen results in a small but statistically significant improvement in survival compared with regimens that do not contain an anthracycline.
As mentioned previously, HFS data indicate that previous episodes of severe hypoglycemia, or negative preconceptions about insulin-based treatment regimens, can significantly influence patient worries and behaviors about hypoglycemia, and fear of hypoglycemia has a significant clinical impact on diabetes management, metabolic control, and long-term health outcomes.
In all mentioned data, P < 0.05.
As already mentioned, data management is an extremely important aspect.
As mentioned in Introduction, neurophysiological data indicate a clear distinction between single neuron-based mirror mechanism and chain-based mirror mechanism in action understanding.
As mentioned above, our unpublished data indicate that it is quite important to choose a suitable time window for PTHrP administration post-injury.
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