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To further examine hippocampal function as it relates to visuo-spatial learning and memory, we performed the Morris water maze task.

Since we observed that the majority of naïve P14 cells that enter the immune response at a later time point remain as memory, we performed a phenotypic analysis in order to examine whether the late transferred cells display a memory precursor phenotype early, by the eighth day after infection with LCMV.

To evaluate our hypothesis that the PKMζ network is bistable and functions as neuronal memory, we performed simulations and bifurcation analyses in the Results section.

To test whether late post-acquisition activity of the aSP13-DA neurons is also necessary for formation of courtship long-term memory, we performed a complementary set of experiments.

As we were specifically interested in the effect of distraction in the formation of source memory, we performed additional ROI analyses on the brain regions where subsequent memory effects were identified from the whole-brain analysis: the left prefrontal cortex, the MTL, and the fusiform cortex [ 56].

DOI: http://dx.doi.org/10.7554/eLife.02736.011 To understand further the functional roles of induced gene expression in the reconsolidation/enhancement of IA memory, we performed correlational analyses on the number of c-fos-positive cells and/or crossover latency among the brain regions of individual mice in the T/R condition.

Similar(54)

To further determine the effect of CDS on the memory retention, we performed the passive avoidance task.

To directly compare recent and remote autobiographical memories, we performed a repeated measure ANOVA.

To investigate further whether correlations between memory-related hippocampus and reward-/punishment-related regions were modulated by the effects of rewards and/or punishments during the successful encoding of IWS memories, we performed additional correlation analyses separately in each condition of Reward, Punishment, and Control during IWS across participants.

Finally, in order to assess whether the reduction in false memories was associated with a reduction of veridical memories (suggesting a failure in memory overall), we performed the same model; but at this time using the veridical memories as the dependent variable.

To further investigate regulation of histone variants following memory acquisition we performed a quantitative proteomics analysis using a nanoLC-MS/MS platform 1 hour after contextual conditioning.

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