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To address this possibility, in Experiment 2 we informed participants of the memory recognition test.
Following the emotional induction, participants performed a memory recognition test.
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Object memory recognition tests suggest that IR+/− mice have an impairment in recognition of familiarized objects; IR+/− mice demonstrate poor performance for both short-term (1 h) and long-term (24 h) memory tests in comparison to that of wild-type mice.
Short term spatial memory recognition was tested by analyzing habituation to the spatial distribution of objects in an open field (novel object exploration task, Fig. 6a).
Supplementary Fig. 1 Effects of Sortilin deficiency on memory deficits: object recognition test.
AAV9-mTERT treated mice from both age groups also showed a trend for improved memory scores (object recognition test) compared to the controls, although they did not reach significance (Supporting Information Fig S4H J).
enhanced memory in the object recognition test regardless of whether animals were exposed to acute stress.
Thus, we tested whether rivastigmine, a widely prescribed reversible AChE inhibitor, was able to improve this deficit, and extended our analysis to include another memory test, the object recognition test.
These investigators suggest that reduced levels of FKBP51 lead to imbalances in glucocorticoid signaling, which, in turn, impair learning and memory in the object recognition test.
We found that overexpression of the NR2B subunit had no effect on CA1-LTD, but rather resulted in enhanced CA1-LTP and improved memory performances in novel object recognition test, spatial water maze, and delayed-to-nonmatch working memory test.
We assessed declarative memory abilities in the object recognition test (ORT) evaluating animal ability to discriminate a new versus a familiar object.
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