The generation of memory cells may depend on Wnt-TCF-1, mTor, and AMPK signaling pathways 27– 27.
Therefore, instead of valence, memory performance may depend on whether rewards are obtained through action or inaction.
Furthermore, as negative results have been often obtained in out-patient samples with rather mild to moderate depression, it may be possible that working memory deficits may depend on symptom severity [ 2] and may only be observable in severely depressed patients.
In other words, the performance of nonlinear systems with memory may also depend on parameters of the input signal, such as its level or its past extrema, as for the hysteretic systems [12].
Whether or not a non-viable vaccine will maintain memory may, therefore, depend on the nature of the adjuvant.
Thus, when a task depends heavily on retrospective memory abilities, performance on the task may depend more on well-documented age declines in retrospective memory than on age declines in prospective memory.
For example, the neuronal suppression to a stimulus may depend on whether memory is associatively evoked or directly activated by a recent presentation of the stimulus.
Hence, the MMN may depend on a memory trace formed by preceding stimuli; i.e., during the presentation of the standard events.
Spoken discourse therefore may depend on auditory short-term memory.
Thus, if embedded into a natural string of events, the net effect of such an adverse life event may depend on the relative strengths of oppositely valenced memories related to its onset (punishment-memory) and its offset (relief-memory).
