Sentence examples for memory lymphocytes and from inspiring English sources

Significant differences were also observed between previously activated memory lymphocytes and naive lymphocytes, indicating a relationship between cell-cycle potential and nanoparticle susceptibility.

Does Schistosoma induce apoptosis or affect the maturation of memory lymphocytes and thus acquire the ability to reinfect its host?

These genes encode the CCL20 chemokine (especially chemotactic for memory lymphocytes and immature dendritic cells), the pro-inflammatory cytokines IL-1β and TNFα, and the IL-12p40 (or IL12β subunit, which is common to the IL-12 and IL-23 cytokines.

Collectively, these findings demonstrate that PP2A also regulates FOXO3a activity in CD8+ memory lymphocytes and indicate that p47phox is necessary for regulating PP2A activity.

It has been demonstrated that inhibiting AKT and IKK reduces FOXO3a phosphorylation in CD4+ memory lymphocytes, and that consequently Bim expression was enhanced and apoptosis was induced in the memory lymphocytes.

We also found that Bim transcription was reduced by 35% in OA-treated p47phox−/− CD8+ memory lymphocytes, and that the percentage of non-viable cells in the OA-treated p47phox−/− CD8+ memory lymphocyte cultures was reduced 30% compared with untreated p47phox−/− CD8+ memory lymphocytes.

Anatomically, the splenic vagus nerve endings are closely in contact with a group of β2 adrenergic receptor- (β2 AR-) expressing T memory lymphocytes (CD4+CD44highCD62Llow) and release norepinephrine (NE), a sympathetic neurotransmitter.

We also found that CD8+ memory lymphocytes from WT and p47phox−/− cultures expressed Bim transcripts and protein.

Interestingly, we found that compared with WT CD8+ memory lymphocytes both AKT and IKK- β were hyper-phosphorylated (pIKK α/ β (ser176/180) and pAKT (Ser473)/pAKT (Thr308)) in p47phox−/− CD8+ memory lymphocytes, which suggest that the dysregulated FOXO3a activity is not mediated by kinase activity alone in p47phox−/− CD8+ memory lymphocytes.

As shown in Figure 4f, the expression of total and phosphorylated Erk 1/2 were equivalent in ex vivo-generated spleen CD8+ memory lymphocytes from WT and p47phox−/− mice, which indicates that the post translational regulation of Bim in p47phox−/− CD8+ memory lymphocytes is comparable to similarly treated WT memory lymphocytes.

The OR and IN administration produced long lasting CD4+ and CD8+ memory lymphocytes that were able to proliferate and produce IFN-γ in response to ZEBOVGP peptides.