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There is growing consensus that human memory is mediated by multiple qualitatively different systems co-evolved to function in a complementary way.
Our findings suggest that recognition memory is mediated by classification of novel exemplars as a match or a mismatch, based on their visual similarity to familiar prototypes.
Human and nonhuman primate data suggest that visuospatial working memory is mediated by a neural network that includes the prefrontal cortex.
The goal of this paper is to elucidate how eyewitness memory is mediated through the exploration of formal conceptualizations of recognition memory and, as a result, be better equipped to evaluate theories such as the relative judgment theory advocated by Wells and colleagues.
We hypothesized that this biological memory is mediated by permanent alterations of the epigenome in stem cell populations, and focused our studies specifically on DNA methylation in CD34+ hematopoietic stem and progenitor cells from cord blood from neonates with intrauterine growth restriction (IUGR) and control subjects.
It has been consistently described that the influence of 5-HT6 receptons on memory is mediated, at least partially, by increased cholinergic neurotransmission.
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Our data thus suggest that prenatal infection-induced deficits in spatial short-term memory are mediated by prenatal maternal effects on the offspring.
To test the hypothesis that the extinction of fear memories is mediated by the proteolytic processing of proBDNF, the synthetic competitive inhibitor of tPA, tPA-STOP (2,7-bis-4 amidino-benzylidene -cycloheptanone-1-dihydochloride 2,7-bis-4 amidino-benzylidene -cycloheptanone-1-dihydochloride 2,7-bis-4 amidino-benzylidene -cycloheptanone-1-dihydochloride 2,7-bis-4 amidino-benzylidene -cycloheptanone-1-dihydochloride 2,7-bis-4 amidino-benzylidene -cycloheptanone-1-dihydochloride 2,7-bis-4 amidino-benzylidene -cycloheptanone-1-dihydochloride 2,7-bis-4 amidino-benzylidene -cycloheptanone-1-dihydochloride
Moreover, the results also show an inverse correlation between levels of the uncleaved precursor of BDNF, proBDNF and the activity of mature BDNF in the CA1 following the extinction of CFM suggesting that extinction of long-term memories is mediated by the processing of BDNF in CA1.
The applied stress increased circulating corticosterone levels as assessed immediately after the retention test, indicating that stress-induced impairment of memory retrieval is mediated, in part, by increased plasma levels of glucocorticoids.
Thus, cocaine-associated memory retrieval is mediated by β-AR activity within the dHipp, and disruption of this activity could prevent cue-induced drug seeking and relapse long after treatment.
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Justyna Jupowicz-Kozak
CEO of Professional Science Editing for Scientists @ prosciediting.com