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Voluntary suppression of a memory is believed to involve prefrontal inhibitory control of the hippocampus where individual memories are processed18.
The algorithm, which imposes less demand on computer time and memory, is believed to be of considerable potential in analyzing a variety of practical problems.
This is not surprising, since working memory is believed to represent a supervisory control system that monitors cognition and self-regulatory behavior (Burgess et al. 2007; Endres et al. 2011; Koechlin et al. 1999; Miller 2000).
Memory is believed to depend on activity-dependent changes in the strength of synapses, e.g. long-term potentiation (LTP) and long-term depression (LTD), which can be determined by the sequence of coincident pre- and postsynaptic activity, respectively.
For example, memory is believed to be ensembles of cells used to store memories.
One could refer to the task in this context as episodic-like, bearing in mind that declarative memory is believed to encompass conscious awareness (Eichenbaum 1999b) and acknowledging the difficulty of precisely ascertaining whether rodents engage in episodic memory formation (Suzuki 2003; Zentall 2006).
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Cognitive deficits in memory, executive function, and working memory are believed to be of major importance to the functional and symptomatic outcome (Fujii and Wylie, 2003; Moritz et al, 2000; Grawe and Levander, 2001; Green et al, 2000) as well as the quality of life (Fujii et al, 2004) of patients with schizophrenia.
Complex brain functions, such as learning and memory, are believed to involve changes in the efficiency of communication between nerve cells.
Cognitive processes such as learning and memory are believed to depend on changes in synaptic efficacy in certain key brain regions, including the hippocampus [23] and the amygdala [24].
Memory may hold the key to time perception, as the clarity of our memories is believed to mould our experience of time.
Successful consolidation of many types of memories is believed to depend on early activation of NMDA-type glutamate receptors, setting into motion a cascade of molecular events that finally leads to durable changes in synaptic properties (Martin 2000; Izquierdo et al. 2006).
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Justyna Jupowicz-Kozak
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