Sentence examples for memory impairment when from inspiring English sources

Previous functional magnetic resonance imaging (fMRI) studies have identified activation in the prefrontal parietal sub-cortical circuit during feigned memory imprefrontal parietal sub-corticalthful telling.

As LTP is thought to be the neural correlate of learning and memory, this would explain why Aβ can produce memory impairment when overproduced [58].

Landrø et al found that only the subgroup of patients with FMS and a lifetime history of major depression showed memory impairment when compared with healthy controls [ 21].

Studies by Shadic and co-workers from 1994 and 1999 found that persons with a history of LB had more musculoskeletal impairment and a higher prevalence of verbal memory impairment when compared with those without a history of LB [ 8, 9].

These oligomers have been shown to be biologically active as they inhibit hippocampal long-term potentiation and create memory impairments when injected into rodents [ 35, 53, 54].

However, during systemic infection or injury (6), this positive regulatory function is disrupted, resulting in acute memory impairments: When inflammation is severe, cognitive impairment may become persistent (7), and when chronic inflammation is present, age-related cognitive impairment is accelerated (8).

The temptation to extrapolate these interesting findings towards real AD is obvious but it requires some caution as other researchers (Balducci et al, 2010) did not observe any protection in prion deficient animals with regard to acute memory impairments when injecting different Aβ oligomer preparations.

Temporal lobe epilepsy (TLE) patients exhibit signs of memory impairments even when seizures are pharmacologically controlled.

First, memory impairment was evident when several objects had to be remembered over short durations, but not when only one item had to be retained.

This could explain why improved memory function was observed in the same mouse model when phenylbutyrate was administered (Ricobaraza et al, 2009) and the fact that SAHA could rescue age-associated memory impairment in mice when injected directly into the hippocampus (Peleg et al, 2010).

Notably, the pattern of results found in Experiment 2 contrast with other RES findings where high rates of suggestibility and memory impairment remain, even when final testing is delayed (e.g., Chan & Langley 2011).