These factors, combined with other possible secondary effects after prolonged use such as psychomotor, cognitive, or memory impairments, limit their long-term applicability.
Some evidence suggests that partial tolerance does develop, and that, "memory impairment is limited to a narrow window within 90 minutes after each dose".
The pattern of memory impairment (impaired recall memory and intact recognition memory) is suggestive of hippocampal damage.
Although, the use of only one test of episodic and semantic memory may limit the investigation of brain correlates of episodic memory impairment in alcoholic subjects, the results found in the present study are consistent with previous reports showing that episodic memory performance is associated with specific brain regions [48], [49].
The multifactorial response of the brain and the complex signaling pathways involved therewith limits the therapeutic efficacy of several antioxidants in ameliorating hypobaric hypoxia induced memory impairment.
Phase 3 involves the analysis of individual diagnostic features (examples include but are not limited to contact sport position, years and level of play, number and severity of individual TBIs, memory impairment, depression, impulsivity, and motor impairment).
To date, pharmacological treatments for dementia have limited effects and there are no known treatments that cure or delay the progression of this memory impairment (Doody et al., 2014; Salloway et al., 2014).
The first is what's called age-associated memory impairment.
Among its toxic effects are memory impairment, which typically begins after just one or two drinks.
Impairment of semantic memory also results in an impairment that resembles a loss of concepts or a language deficit more than it resembles a memory impairment.
