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Verbal memory impairment has been well explored in schizophrenia, but it is unclear whether findings relate to the type of material to be learned or the component process required by the memory task.
Though oxidative stress contributes to neuronal degeneration and apoptosis in hypobaric hypoxia, the ability for phenylethanoid glycosides of Pedicularis muscicola Maxim (PhGs) to reverse high altitude memory impairment has not been studied.
Memory impairment has been found in schizophrenia and appears to be responsible for the characteristic symptoms of the disorder [ 108].
In particular, memory impairment has been associated with high-dose or high-potency benzodiazepines, particularly in older people [ 102, 107].
Frontal dysfunction in MHE patients, such as attention [ 19, 21] and working memory impairment, has been consistently reported [ 34].
14, 15 Memory impairment has figured more significantly in some studies than others 9, 10 but is a less prominent feature than in other dementing illnesses.
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Brain lesions associated with memory impairment have also been found here.
Similarly, aspects of memory impairment have been reproduced.
The memory impairment had lasted less than a year in 8%.
Moreover, and in agreement with our findings, neuropsychological deficits (including memory impairment) have been associated with hypoperfusion within the frontal cortex and basal ganglia [ 18, 19].
Approximately one-half of individuals with amnestic MCI [ 25, 29], characterized by memory impairment, have elevated Aβ on imaging and have an increased risk of conversion to AD (see below).
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CEO of Professional Science Editing for Scientists @ prosciediting.com