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The neuropharmacological mechanisms involved in MDMA-induced memory impairment are not clear.
The results suggest that neural correlates of feigned memory impairment are distinguishable from answering randomly and answering incorrectly in healthy subjects.
In patients with schizophrenia, specific deficits, such as memory impairment, are reliable predictors of function [3], and are considered critical targets for the development of novel pharmacological or psychosocial therapies [4].
Clinical implications of memory impairment are discussed in further detail below.
Our results suggest odds of late-life severe memory impairment are substantially increased in middle-aged adults with a history of AUDs.
Specifically, MTL atrophy and associated episodic memory impairment are hallmark features of AD, and both progressively decline over the course of the disease [ 2, 3].
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The previously found impaired spatial memory impairment was not replicated (Fig. 5D; P>0.7).
While acute cortisol administration has been found to impair retrieval of emotional memories in healthy subjects, the duration of this memory impairment is still unknown.
The Mayo Clinic doctors studied these patients and discovered that their memory impairment was very specific -- their only problem was remembering experiences of the last few minutes or hours or days.
Memory impairment is an early-delayed effect of radiotherapy (RT).
After 30 min, memory impairment was induced by administering scopolamine (1.5 mg/kg, i.p).
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