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Implicit auditory memory formation has been documented under general anesthesia.
A beneficial effect of sleep after learning, compared to wakefulness, on memory formation has been shown in many studies using a variety of tasks.
Given that short time window and accounting for the time it takes the drug to become active in the brain, the first new protein synthesis required for context memory formation has a rapid onset, and is transient, Xu says.
While prior studies in mood-disorder patients point to the medial temporal lobe in the genesis of mood-congruent memory (MCM) bias, the interaction between mood and emotional memory formation has not been investigated in healthy participants.
Memory formation has multiple temporal phases that engage specific molecular, cellular and systems mechanisms [1], [2].
These general laws hold for many species, and the neurobiological basis linking reward processing and memory formation has been investigated with both electrophysiological and neuroimaging methods.
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Sub-optimal levels of hippocampal activity are commonly attributed to unsuccessful memory formation, whereas the supra-optimal levels of hippocampal activity related to unsuccessful memory formation have been rarely studied.
DOI: http://dx.doi.org/10.7554/eLife.01982.002 The mechanisms of memory formation have been the subject of considerable study (Morris et al., 1988; Kandel, 2001).
DOI: http://dx.doi.org/10.7554/eLife.07578.001 Investigations of human memory formation have focused chiefly on the roles of the hippocampus and the neocortex.
Genes involved in the process of memory formation have been studied intensively in model organisms; however, little is known about the mechanisms that are responsible for natural variation in memory dynamics.
While the cytokines, surface molecules, and signaling components involved in T cell memory formation have been extensively studied, the molecular pathways supporting these cell fate decisions are poorly understood.
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