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The delay-related NIRS data for false memory errors was examined.
In Experiment 2, the reference memory task, mice also showed significant learning in both mazes and the rate of reduction of reference memory errors was the same.
The number of reference memory errors was also increased significantly in diabetic rats compared with control (P < 0.05), suggesting that learning and memory ability in the diabetic rats were damaged by day 71 (Table 3).> -wrap-foot> A rat diabetes model (streptozotocin-treated group, n = 15) is compared with control (n = 10).
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Causal inferential memory errors were most pronounced among 5- and 6-year-olds.
Working memory errors were indicated by re-visiting an already visited arm during a trial, whereas visiting an arm that was never baited on any trial indicated a reference memory error.
In eight-arm radial maze experiments, both reference memory errors and working memory errors were significantly decreased in mice by fractions 1, 2 and 4. In addition, these fractions were also effective in promoting memory in the passive avoidance test in mice and rats.
However, matching overall accuracy alone may not be sufficient if different types of memory errors are elicited via different mechanisms in schizophrenia patients.
These memory errors were well accounted for by modeling memory as a noisy but unbiased version of perception constrained by the matching methods.
Search errors and memory errors were low (Exp. 1A, 1.25 % and 4.86 %, respectively; Exp. 1B, 1.43 % and 3.99 %, respectively) and were removed from the main analyses.
Working memory errors are scored in this task as revisits to "incorrect" holes which subjects have already investigated within a probe trial.
It is possible that memory errors are an inevitable by-product of our adaptive memories and that semantic false memories are specifically connected to our ability to learn rules and concepts and to classify objects by category memberships.
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Justyna Jupowicz-Kozak
CEO of Professional Science Editing for Scientists @ prosciediting.com