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Earlier this year, Yu-Chin Chiu and Tobias Egner of Duke University in North Carolina reported that response inhibition impairs memory encoding.
This corresponded to activation increases in the right parahippocampal gyrus during memory encoding.
Glucose administration increased activation in brain regions associated with successful episodic memory encoding.
Therefore, beta oscillations are suited for tACS during verbal memory encoding.
Left lateral prefrontal cortex (PFC) is consistently activated in neuroimaging studies of memory encoding.
These structures support critical components of verbal memory, including working memory, encoding, and retrieval.
By doing so, we aimed to better answer whether cuing event structure per se benefited memory encoding.
This suggests that anterior MTL structures underlie memory encoding as judged by subsequent memory effects, and that more posterior activity detected in other fMRI studies is related to deficiencies of blocked designs in the analysis of memory encoding.
Lesion-deficit studies have provided evidence for a functional dissociation between the left medial temporal lobe (MTL) mediating verbal memory encoding and right MTL mediating non-verbal memory encoding.
It is well documented that acute administration of the benzodiazepine hypnotic drug triazolam (Halcion®) impairs episodic memory encoding.
Optimizing sensitivity in such paradigms is critical for the reliable detection of regions most closely engaged in memory encoding.
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Justyna Jupowicz-Kozak
CEO of Professional Science Editing for Scientists @ prosciediting.com