Sentence examples for memory dysfunctions and from inspiring English sources

This assumption is supported by learning and memory dysfunctions and aberrant regeneration in monkeys exposed to Ecstasy compounds [8], [34].

Our study shows that although the level of PGI2 and ratio of PGI2/TXA2 in the hippocampus were significantly induced, the rats displayed obvious spatial learning and memory dysfunctions and obvious hippocampus neurons damage, probably because the increased PGI2 level and PGI2/TXA2 ratio were insufficient to antagonize the I/R-caused damage.

The present study suggests that the L-arginine-NO signaling pathway is involved in the protective effect of hesperidin against cerebral I/R-induced memory dysfunction and biochemical alterations in rats.

Alcoholics often have impairments of anterograde memory, including spatial memory dysfunction, and a tendency toward response perseveration.

The second is a semantic memory subtype, due to verbal memory dysfunction and characterized by errors in the retrieval of arithmetic facts.

Memory consolidation is also of considerable clinical interest in the treatment of both memory dysfunction and anxiety disorders such as posttraumatic stress disorder that may be based on maladaptive learning processes.

Therefore, it is less likely that subjective memory complaint has served as a strong factor biasing the findings of an association of memory dysfunction and APOE ε4 in the present study.

Working memory dysfunction, and executive function deficits more broadly, have been found across a number of psychopathologies, including attention deficit disorder, autistic spectrum disorder, traumatic brain injury, Alzheimer's disease, schizophrenia and depression.

While Aβ deposition is a hallmark of AD pathology, hippocampal atrophy is a common feature of AD that correlates well with episodic memory dysfunction and has emerged as a biomarker for this condition.

The role of heparin coating in CPB as a tool to prevent memory dysfunction and neurological deviations is not conclusive, despite a less favourable outcome in terms of S100 release and subjective rating of memory function for patients in the Baxter uncoated control group.

The authors suggested that high Aβ42 levels and aggregates are not sufficient to induce memory dysfunction, and that derivatives of APP processing (which were obviously not generated from the Bri2-Aβ construct used) are contributing to the toxicity seen in APP transgenic mouse models (Kim et al., 2013).