Sentence examples for memory deficit after from inspiring English sources

The results of this study showed that liothyronine can prevent memory deficit after ECT in patients with MDD.

We also suggest to conduct a study on patients with MDD who receive liothyronine for preventing cognitive effects of ECT but still suffer from symptoms of memory deficit after ECT to find predictive characteristics of effectiveness of such treatment.

The findings support the possibility that the auditory working memory deficit after MTL ablation is due to transection of downstream auditory projections, and indicate that the candidate structures for mediating auditory working memory are the ventral medial prefrontal cortical areas, the medial thalamus, or both.

Memory deficits after unilateral inactivation of the ventral hippocampus or amygdala prevent us from determining whether the mPFC functionally interacts with the medial temporal lobe using a disconnection approach.

Several efforts have been made to find a practical and effective preventive treatment for memory deficits after ECT.

Hamidia et al. [ 21] also reported that liothyronine could prevent memory deficits after ECT in patients with MDD.

In our study, anti-inflammatory drug treatment reduced the proliferation and/or activation of microglia/macrophages, increased hippocampal neurogenesis and rescued the spatial memory deficits after ICH.

The involvement of inflammation and microglia activation in spatial memory deficits after striatum ICH could be supported by some indirect evidence.

An increased Aβ42/Aβ40 ratio enhanced aggregation and neurotoxicity in vitro and caused memory deficits after a single intraventricular injection in wild-type mice [ 44].

There may be underreporting especially of early mobile phone use because of memory deficits after surgery, but most concerns have been raised for a potential distortion in reporting side of the head the phone is held during calls.

Two models have been discussed to explain memory deficits after ATLR: the hippocampal reserve theory suggests that it is the capacity of the contralateral hippocampus which preserves postoperative memory function whereas the functional adequacy model suggests the functional reserve of the ipsilateral hippocampus as the key structure to support memory function after surgery (Chelune et al., 1991).