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Furthermore, the design enabled us to reduce potential differences in memory strength between item and associative memory by increasing task difficulty in the item recognition memory test.

The researchers suggest that caffeine blocks consolidation of new material into long-term memory by increasing levels of a neurotransmitter called acetylcholine in the hippocampus (the same neurotransmitter that naturally decreases during slow wave sleep).

They also have an important role in learning and memory by increasing the density of dendritic spines in hippocampal pyramidal cells and enhancing excitability and synaptic plasticity (Hao et al. 2006; Prange-Kiel and Rune 2006).

Our results suggest that the present task facilitated the use of a feature strategy possibly through increasing the salience of the feature cues or through taxing memory by increasing the number of search locations.

Green tea also contains catechin, and chronic treatment improves spatial learning and memory by increasing PSD95, BDNF, and CaMKII while suppressing A β (1 42) level via the PKA/CREB pathway in the hippocampus of learning-and-memory-impaired SAMP8 mice [ 112].

A significant increase in dendritic intersections, branching points, and dendritic processes arising from the soma of neurons in the amygdale region in CT-treated rats was observed in comparison with age-matched saline controls, suggesting that CT enhances memory by increasing the functional growth of neurons [ 65].

Similar(54)

Time sharing between different tasks running on the processor allowed further optimization in the memory system by increasing the memory reuse option.

A second series of experiments that used a similar design was intended to induce differences in the nature of memory traces by increasing the time allowed for encoding the to-be-remembered items.

In Swedish mutant APP transgenic mice (APP23), long-term hypoxia has been shown to markedly increase Aβ deposition and neuritic plaque formation and potentiate the memory deficit by increasing β-site APP cleaving enzyme 1 (BACE1) gene transcription and expression, primarily mediated by the binding of hypoxia-inducible factor-1α to the BACE1 promoter [25], [26].

In addition, EE rescued sevoflurane-induced neurotoxicity and learning and memory impairment by increasing PPAR- γ levels in the hippocampus.

In previous studies, we found that sevoflurane induces neurotoxicity and learning and memory impairment by increasing neuroinflammation and inhibiting synaptic function [ 7].

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