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Integrating advances in lipid membrane research has enabled therapeutic development.
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Membrane fouling research has become a focus, significant amount of work has been dedicated to membrane surface modification for improving anti-fouling performance.
The mechanism by which dendrimers inhibit P-gp is via endocytosis; the dendrimer passes into the cell interior; this inhibition process could be due to cell membrane alterations and ATPase inhibition; research has shown that P-gp substrates that are encapsulated within dendrimers show an increased intracellular concentration [ 61].
Up until now the focus of TLR signalling research has been on delineating the membrane-proximal adaptor molecules utilised.
Most of the deposition research has been done in porous alumina membranes [8 10].
A considerable amount of material research has investigated crosslinking of dense film membranes to increase plasticization resistance.
Recent research has been directed toward synthesis of zeolite membranes by more efficient methods or on more cost-effective supports and microstructural engineering of the zeolite membranes.
However, scarce research has been detected in literature regarding end-of-life membrane management.
Recent research has identified several cryptic fragments of extracellular matrix/vascular basement membrane proteins that have potent antiangiogenic properties in vivo.
Our research has instead focused on the rapid, nongenomic or membrane-initiated effects of these environmental contaminants.
Recent research has focused on the use of continuous reactor systems such as membrane, fixed bed and microreactors.
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