Sentence examples for membrane potential resulting from inspiring English sources

It has been previously shown that even when phage and bacterial lysins are deleted, phage induction decreases cell viability as phages express holins that collapse the cell membrane potential resulting in host cell death [32].

CCCP dissipates the mitochondrial membrane potential, resulting in recruitment of Parkin to mitochondria and Parkin-dependent mitophagy (8).

It is known that increase in intracellular calcium might be one of the factors responsible for disrupting the mitochondrial membrane potential resulting finally into cell apoptosis [ 36- 39].

It is traditional view that the activation of KATP channels shortens action membrane potential resulting in decreased influx of Ca2+ and prevention of Ca2+ overload [30].

The intrinsic apoptotic pathway involves the loss of mitochondrial membrane potential resulting in cytochrome c release and subsequent caspase activation [ 9].

In the late 1990s, decreased mitochondrial membrane potential resulting in an increase in cytosolic calcium was one mechanism elucidated by which loss of mitochondrial DNA activates signaling pathways [ 6].

In cultured embryonic chick heart cells, alterations of extracellular Na (Nao) and Ca (Cao), intracellular Na (Nai) and Ca, extracellular pH, and membrane potential resulted in changes in Na and Ca contents that were consistent with sarcolemmal Na-Ca exchange.

In addition, loss of membrane potential resulted in loss of bio-transformation, and this may suggest that this targeting approach may be less useful in the presence of pathological mitochondrial dysfunction.

The relationship between the minimum and the steady-state membrane potentials resulting from a hyperpolarizing current is similarly ambiguous.

Taken together, these results implicate that ANT2 shRNA changes the Bcl-2 family balance in mitochondrial membranes, favoring a proapoptotic pore-forming status, and thereby causes the disruption of mitochondrial membrane potentials resulting in cell apoptosis.

99mTc-MIBI, a lipophilic cationic myocardial perfusion agent, is considered to be retained in the mitochondria by the higher membrane potential, and the loss of mitochondrial membrane potential results in rapid washout of 99mTc-MIBI from the myocyte [14].