Your English writing platform
Discover LudwigSuggestions(2)
Exact(3)
The negative sign of the membrane potential indicated that the inner surface of the cell membrane is relatively lower than the immediate exterior surface of cell membrane [ 69].
Subsequent studies, using valinomycin to manipulate membrane potential, indicated that hyperpolarization increased the susceptibility to blockade by ryanodine of the Ca2+ release evoked by Ni2+(182).
At 24 h post-treatment, both MM.1S and 8226 cell lines showed a drop in mitochondrial membrane potential (indicated by a decrease in the ratio of red signal to the green signal) with irradiation, which was significantly inhibited by pre-treatment with ICCM or IL-6.
Similar(57)
PABA addition resulted in negligible effects on plasma membrane potential indicating the involvement of the NO-generating capacity of PABA/NO, its initiation of Ca2+ fluxes, and secondary eNOS-mediated intracellular NO production.
A decreased mitochondrial membrane potential indicates early apoptosis.
Mitochondria retained their membrane potential indicating, as discussed before [ 12, 15], that the protein was correctly assembled in the OMM.
To confirm that these single-molecules were behaving normally, traces from many different fluorescent spots were summed together; these summed traces recapitulated the macroscopic response of ArcLight' to changes in membrane potential indicating that their function was uncompromised.
This phenotype requires both Parkin and PINK1 expression and the loss of the inner mitochondrial membrane potential, indicating that it occurs upon mitophagy induction downstream of PINK1-induced Parkin toanslocation to mitochondria.
In addition, while parkin KO mature mouse cortical neurons or MUL1 knockdown neurons show only mild neuronal phenotypes, neurons with parkin KO and MUL1 KD show significantly diminished mitochondrial membrane potential, indicating mitochondrial dysfunction.
Neither 100 nor 200 μmol/L of the STAT3-specific inhibitor Stattic affected mitochondrial autofluorescence or membrane potential, indicating that Stattic had no acute toxic effect on mitochondria (Appendix Fig. 12).
Both substitutions rendered the resulting mutant channels insensitive to inhibitory (0 mV) and activating (−100 mV) membrane potentials, indicating that both changes eliminated the influence of Ct on function.
Write better and faster with AI suggestions while staying true to your unique style.
Since I tried Ludwig back in 2017, I have been constantly using it in both editing and translation. Ever since, I suggest it to my translators at ProSciEditing.

Justyna Jupowicz-Kozak
CEO of Professional Science Editing for Scientists @ prosciediting.com