Sentence examples for membrane potential increases from inspiring English sources

Mitochondrial membrane potential was evaluated with the lipophilic cationic probe JC-1 (Invitrogen), which changes reversibly its color from green to orange as the membrane potential increases [24].

The JC1 dye undergoes a reversible change in fluorescence emission from green to red as mitochondrial membrane potential increases.

JC-1 is a lipophilic, cationic dye that can selectively enter into mitochondria and reversibly change color from green to red as the membrane potential increases.

In addition, our study has shown that Ang II induces mitochondrial KATP channels to open, depolarizes mitochondrial membrane potential, increases ROS generation by the mitochondria, and mediates the downstream signal transduction in VSMC [ 18].

JC-1 (5,5',6,6'-tetrachloro-1,1',3,3'-tetraethylbenzimidazolo carbocyanine iodide) [ 14, 15] is a lypophilic, cationic dye that can selectively enter mitochondria and reversibly change colour from green to red as the membrane potential increases.

Rapid calcium fluxes (within 30 s), loss of mitochondrial membrane potential, increases in reactive oxygen species (from 6 h after medium transfer), an increase in the number of apoptotic cells (48 hours after medium transfer) and a marked reduction in clonogenic survival (after 9 days) were observed.

CCI injuries also resulted in an immediate decrease in ATP concentrations, decreased mitochondrial membrane potential, increased mitochondrial permeability transition and increased ROS production in isolated cortical synaptosomes (Sullivan et al., 1998; Sullivan et al., 1999).

Methods for cytotoxicity analysis showed that PEG-AuNP did not affect cell viability, but intracellular ATP levels and mitochondrial membrane potential increased.

Apoptosis was confirmed using reactive oxygen species that triggered a decrease in mitochondria membrane potential, increase in Cyt-C release, activation of caspases 3 and caspases9 and increase in the ratio of Bax/Bcl-2.

Importantly, myocardial and mitochondrial ultrastructural abnormalities were more pronounced in HF-fed offspring with prenatal fat exposure, shown as loss of mitochondrial density and membrane potential, increased ROS generation and apoptosis.

Our results show that rapamycin can improve mitochondrial membrane potential, increase mitochondrial mass and reduce ROS production.