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Notably, Pfister et al. [37] consider synapses inferring presynaptic membrane potential given only spike observations.
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Quantification of cell shrinkage and of depolarisation of the mitochondrial membrane potential gave similar results (Fig 13c white and grey bars, respectively).
Membrane potentials given were not corrected for liquid junction potentials.
The bursting oscillations of membrane action potential give rise to oscillations in the intracellular calcium concentration and in the secretion of insulin into blood stream.
This finding is consistent with our previous work [29] reporting that addition of reagents known to decrease the membrane dipole potential, give rise to di-8-ANEPPS difference spectra of similar profiles to those reported here.
Let us consider a non-connected (QIF) neuron, i.e., its membrane potential is given by { d d t v ( t ) = v 2 ( t ) + I b If v = + ∞ then v = − ∞. (11).
The K+ channel conductance dependence on the membrane potential is given by: (36) ν K + ψ = exp α K + ⋅ ψ + d K + λ K + + exp α K + ⋅ ψ + d K +.
The relationship between dimensional and non-dimensional quantities for Ca2+ concentration and membrane potential are given in Table 2. NVK, ANG, BG, MZQC, ID, CH and AA developed and implemented the project under the supervision of DGB, RNK, YU and NVV.
This quickly decreases the cell membrane potential and gives telavancin its rapid concentration-dependent bactericidal activity.
Finally, as we did for (overline{J}_{ij} (t )), we suppose that the external input current is deterministic (if we interpret (mathscr{B}_{i} (t )) as the background noise of the membrane potentials) and given by I_{i} (t )=I_{i}^{c}+ sigma_{4}I_{i}^{v} (t ) (2.9) where (I_{i}^{c}) is constant in time, while (I_{i}^{v} (t )) is variable.
These membrane potentials are given by eqs.
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