Sentence examples for membrane of type from inspiring English sources

Hypochloremia reduces chloride reabsoprtion via the SLC26A4 (pendrin) encoded Cl-/HCO3 exchanger that is positioned on the apical membrane of type B intercalated cells [ 10].

Normally the V-ATPase is translocated from the cytoplasm to the apical plasma membrane of type A-ICs upon acidosis, whereas it is translocated from the basolateral plasma membrane to the cytoplasm in type B-ICs (Schwartz et al, 1985).

They include SLC26A6 in the proximal tubule that mediates apical chloride-anion exchange, and SLC26A4 (pendrin) in the distal nephron that mediates chloride-anion exchange across the luminal membrane of type B intercalated cells.

As previously reported, when using the 1B1 antibody that detects all Hts isoforms except for ShAdd, Hts immunoreactivity is found predominantly at the postsynaptic membrane of type Ib and Is boutons, with lower levels found throughout the muscle (Fig. 1A) (Petrella et al., 2007; Pielage et al., 2011; Wang et al., 2011; Zaccai and Lipshitz, 1996).

Examples are SLC4A1 (also known as AE1), which mediates chloride-bicarbonate exchange in the basolateral membrane of type A intercalated cells (exchange of chloride into intercalated cells and bicarbonate into plasma), and SLC4A4 (also known as NBC1), which mediates sodium-bicarbonate co-transport in renal proximal tubule cells [ 46, 48].

This sarcoplasmic localization is thought to increase the presence of reactive oxygen and nitrogen species (ROS/RNS) [ 47], and the present observation of alterations in intermyofibrillar network and a subsarcolemmal increase and/or clustering of mitochondria along the fiber membrane of type I fibers further supports this notion as mitochondria are a major source of ROS [ 57].

Recent studies have shown that contact of lung tissue with NPs about 50 nm in size leads to perforation of the membranes of type I alveolar cells and the resultant entry of the NPs into the cells.

It is less likely that Baf A1 affects cytoplasmic pH in type II cells since V-ATPase is highly enriched in lamellar bodies and there is only minimal expression of V-ATPase on plasma membranes of type II cells.

Thiamine transporter protein contents of erythrocyte membranes of type 1 and type 2 diabetic patients were increased.

TIMP-1 (tissue inhibitor of metallopeptidase1) expression is regulated through STAT3 [ 38] and TIMP-1 is induced in the epiretinal membranes of Type 1 and Type 2 diabetic patients [ 39].

These are the outer membrane subunits of Type II and Type III secretion systems.