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The most frequent complications were bleeding (45 %) and membrane failure (18%%).
For each composition studied, membrane failure occurred at uniaxial extension ratios of 3.8.
Membrane failure, defined by a threshold convective hydrogen leak rate, is obtained after 160 h of operation.
This settling leads to microstructural changes and pore formation on the feed surface of the membrane film that ultimately results in membrane failure.
Compounded chemical and mechanical degradation effects are therefore considered particularly harmful, given the ability to bridge mesoscale morphological alterations induced by chemical degradation to macroscale damage propagation under mechanical stress cycles, leading to ultimate membrane failure.
The breaking speed predicted by the 3D model based on maximum principal stress criterion for a single fiber is between26%% (membrane failure) and 76% (combined membrane and flexure failure) less than the 1D analytical solution.
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Membrane failures were evaluated by field-emission scanning electron microscopy (FE-SEM).
The clinical signs of zinc toxicosis have been reported as vomiting, diarrhea, bloody urine, icterus (yellow mucus membrane), liver failure, kidney failure and anemia (Fosmire 2001).
Finally by neglecting the plastic deformation of the membrane, its failure stress, Neo-Hookean and Mooney-Rivlin parameters are obtained.
To examine if the separation of epidermis from the basement membrane, and failure of keratinocytes to adhere to culture plates was related to the expression of integrins, we studied the expression of beta 1 integrin in tissue sections (Figure S7A).
According to Smyth et al. [ 29], null mutation of LAMC1 causes embryonic lethality because of the absence of the basement membrane and failure to differentiate the endoderm.
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